Drug resistant Candida is pointed out to be the number one killer, sudden heart attacks in this report - which also explains how drug resistant bacteria initialize cancers and tumors!
Most suddenly occuring heart attacks are caused by Drug Resistant Candida, moving into the blood stream from an infected tooth. Cancers, tumors, and many of the "gene-related diseases" are caused by the DNA modules (i. e., plasmid DNA) that has become active after being passed into a given cell, from an unseen, drug-resistant bacteria. These findings were achieved by way of an extensive, epistemological study.
(PRWEB) November 6, 2003
(PRWEB0 November 4 2003--This is a two part report on drug resistant bacteria, as the hidden cause of heart attacks, strokes, cancers, tumors and other gene related diseases. I believe all these diseases are explained in this brief essay, and this information needs to be somehow placed on the Internet, and in front of the rest of the world where it might be read by someone like BjÃ¶rn NordenstrÃ¶m, M. D., Ph. D., (former chairman of the Department of Radiology at Karolinska Hospital and chairman of the selection committee that picks the Nobel Laureate for Physiology of Medicine.)
Part I. These facts explains how drugs and various DNA transferring bacteria act as the major cause of heart attacks, strokes, and many cancers, gene related diseases. Primarily Â any of these diseases are known to emerge following exposure to a chemical, carcinogen, drug, or sometimes, even after eating a certain food which induces an upset of the normal ecological balance of the intestinal flora. In reality, heavy smoking, or any of the above can commonly results in a general suppression of the hosts immune system.
First, the data on drug-resistant Candida and how it happens to be the number one killer, in that, it causes a great majority of the unexpected heart attacks and strokes. As you may already know, exposure to various environmental toxins often causes people to become susceptible to Candida; the organism wasnÂt even known until the early 1990Âs, and it is still being ignored by most individuals in the medical field because they simply donÂt know what to do to stop it. Frankly, chemicals, food additives, and poor nutrition enable Candida to thrive. This type of bacteria is also able to transform itself, growing root-like, rhizoid like legs and producing toxins that further lower a personÂs immunity. In other words there are many things that can (and usually do) result in Candida overgrowth. More specifically, cortisone, prednisone, and other steroid-type drugs are known to cause the opportunistic Candida to thrive. There are times when these drugs, and other drugs and chemicals, paralyze the bodyÂs defense systems, just as birth control pills and various estrogens are known to upset hormonal balances within the body, and thereby these factors also contribute to the abnormal growth of excessive Candida. (Note: some bacteria in the Clostridium family also break down fats into the minor estrogens, otherwise known as Âimpeded estrogens.Â)
There are also various contributing factors such as the western diet being typically high in sugar and other refined carbohydrates; that alone has already produced an entire generation of overweight people. The excessive weight is largely due to the growth of drug-resistant Candida and other high-energy, drug-resistant bacterium. The Candida in particular, are capable of producing so many toxins that they simply overwhelm the adrenal gland operations. The result is Âno energyÂ or Âlow energyÂ people, who can only find temporary relief from the Candida, by frequently stuffing themselves with all kinds of food! Candida are also known to Âimpair immune functioning by directly and negatively impacting the helper-suppresser ratio of T lymphocytes,Â (according to Charles R. Caulfield in his article "Natural Therapies For Candidiasis InfectionsÂ in the San Francisco Sentinel, August 27, 1992.)
Everyone should be made aware of the fact that when it begins to emerges in ever increasing numbers in your body, this organism (Candida) progressively spreads out from the lower bowel to colonize the entire digestive tract, successively including the stomach, (especially in cases of low or no stomach acid) and from there it will spread into the throat, mouth, and even the nasal passages. It can even get into your eyes. When it does, it can cause blindness to occur. Once it moves into the throat and/or mouth, these ultra tiny microorganisms may even move down into the lungs. They are drug resistant, meaning, they can exchange DNA so upon reaching the lungs they emerge as Candida spp. a frequent asymptomatic colonizer of the upper respiratory tract, and especially well know for their ability to emerge among hospitalized patients. Or, these same microbes may emerge as primary Candida pneumonia, Âlife-threatening in patients with cancer since it directly contributed to the death of 84% of the patients,Â in one study of a series of patients. (Medicine (Baltimore), 1993 May;72(3):137-42; PMID: 8502166.)
The all important part for you to note - in those individuals where an infected tooth or periodontal disease is present, the candida may move into the mouth and then, directly into the blood stream. Then the Candida readily attached to the plaque deposits in the arteries of the heart - this is how it often causes a fatal heart attack! As soon as it transforms and begins to put down its budding rhizoids, the finger-like roots break up plaque deposits that have been building up over the years and years. That causes a rapidly forming mass in a given artery, and most persons are overwhelmed by the rapidly occurring injury that results. I believe this rather clearly and simply explains why the incidence of heart disease is about twice as high in people with periodontal (gum) disease. It also explains how years and years of plaque build up, can be quite suddenly acted upon, and subsequently, in only a day or two it is quite readily "broken up" inside an artery. Until this explanation came along, however, no plausible cause had ever been suggested. Other recent studies have indicated however, that the most common strain of bacteria in dental plaque also causes blood clots. So the all important thing is the rhizoid forming Candida that escape into the blood stream, and not the blood clots around a tooth.
The big "missing factor" in all this is Co enzyme Q10. If everyone with dental problems was actually taking Co enzyme Q10, they wouldn't have "bleeding gums." Q10 shuts down all periodontal diseases and it rapidly strengthens the heart at the same time. When my own father had a heart attack in 1985 or so, 90% of those heart patients consistently died within a month as a result of the congestive heart failure that frequently develops. I put him on Co enzyme Q10, AM and with lunch. In less than 30 days his heart was back to absolutely normal, 100% restored! The average recovery rate, right now, for patients who have congestive heart problems - and they are taking Co enzyme Q10 - is very close to 100% in a time span of only 30 days! Unfortunately, if you're one of those persons whose doctor doesn't know about Coenzyme Q10 and the heart - you probably won't live long enough for him to tell you about it, unless you order it and start taking it on your own! Likewise, if you've got an infected tooth or periodontal diseases, along with Candida, the plain facts are that the chances go up and up for you getting a heart attack, each time you take a much needed nap, or lie down to sleep, simply because that's when the Candida are able to move up, more readily. Incidentally, many persons experience their first heart attack, immediately following their being confined to bed for a week or so, such as immediately following a cold. Medical researchers have been recently searching for various antigenic factors coming up, just prior to the heart attacks, and they have even concluded that having the flu bug, may frequently lead to heart attacks! In contrast, the probability is far, far greater, that the most common cause of heart attacks is still those blasted little, drug-resistant Candida - simply because are being prompted to grow and grow at an even far greater rate(!) when any given patient is given almost any form of flu fighting antibiotic drugs!
The unseen relationship between increased heart attacks, candida and an infected tooth or periodontal diseases and other heart illnesses is also neatly explained in terms of the overt facts of history. In the not too distant past, the lessons of medical history show that while no one in previous generations had the ability to explain how great numbers of candida were what caused the toxins of Âsevere heartburn,Â everyone was nevertheless were very much aware that Âsevere heart burnÂ frequently graduated to - Âfatal heartburn!Â Other researchers have also found that similar infections with Cytomegalovirus (CMV), (a usually dormant virus that can cause mononucleosis,) is associated with a higher risk of heart attacks or strokes. As I have mentioned, some claim it's because of the flu virus. Other have found that infections with more than one of several common disease-causing germs can also increase the risk for heart attacks and strokes. They think people who had one or more of CMV, Chlamydia pneumonia (respiratory infections), Helicobacter pylori (ulcers), and hepatitis A (a liver disease,) were also at increased risk for heart attacks. Thus, people exposed to all four pathogens had a higher risk compared with those exposed to just one of the four! I must still contend that no one previously grasped the fact that heart attacks and strokes are mainly due to the sudden presence of drug resistant Candida - with its ability to transform into a cell complete with rhizoids, after entering into the blood stream. And the Candida is obviously allowed (or caused) to readily build up to tremendous proportions whenever patients are given almost any form of antibiotics or almost any other medications for any of the above! (By the way, are you aware that Helicobacter pylori are the pathogens most often found to accompany Candida, in those persons where overwhelming Candida has been found to be present!)
Part II. This leads directly to another highly debated factor of these diseases, or what is known as Âstress factors.Â This requires you can disconnect for a moment, from thinking about stress in terms of having Candida and H. pylori in your stomach, and they are eating you alive, while everyone is telling you "it's all in your head!" I mean, in addition to that type of Âpsychological stress,Â or "bodily stress," I believe I can explain to you an infinitely more harmful type of stress that I see as being truly at work as part of the cause, in many other diseases. What I mean by this is that every tissue has its own set of ecological values - which match those of the environment required by some type of nearly invisible bacteria. Furthermore, when the microbes involved are high-energy, drug resistant bacteria, in those cases where infection occurs, they grow in abundance and they commonly generate their own toxic byproducts - as does Candida, H. pylori, and many other organisms. In other words the presence of these microbes at a cellular level is what truly produces the so-called Âstress factor,Â that no one has been able to come to grips with, or fully understand. Unfortunately, this is the way in which the knowledge of modern medicine grows. One day, everyone finally beings to understand Âthe real cause of stressÂ Â and how or why it emerges at a basic cellular level, or at a basic "tissue level," and all of a sudden everybody begins to realize that regardless of what tissue may become Âinfected,Â or cancerous, itÂs a very common bacteriological mechanism!
Why doesn't everyone know about all this? The answer is simply because no one has ever previously realized that all the "stress originating" cancers and the other Âdrug resistant diseasesÂ can be viewed as a class - once one understands how they are caused by a specific class of very small bacteria which are able to transfer DNA into a living cell. These findings were achieved through a great deal of study, and patient epistemological investigation. Another real part of the overall problem has always been that these "stress causing" microorganisms are in fact, very difficult to see and seemingly impossible to isolate, because: a) the drug resistant strains are of an incredibly small in size, and the transferable DNAÂs that they exchange among themselves, or with other cells, are in fact, far, far smaller; b) typically, they are able to leave their ultra tiny, transferable DNA packets behind (now known as DNA plasmids) Â all of which is readily proven by way of the initial controversy surrounding the discovery that they can only be seen by way of using an electron microscope accompanied by Âmetal staining techniques;Â c) the presence of the unseen yet transferable DNA packets, (identifiable only by using an electron microscope and metal staining techniques,) actually explains why cancers and a large group of other drug resistant diseases reoccur so rapidly; d) the involvement of transferable packets of DNA modules also explains why this class of diseases (i. e., Cancers and The DNA Transfer Diseases,) are so difficult to medicate or otherwise eliminate; e) the drug-resistant bacteria that causes The DNA Transfer Diseases, typically grow very slowly under laboratory conditions, i. e., far to slowly to be readily recognized and/or discovered by using ordinary laboratory techniques; f) these organisms can change or shift from one phase to another when being isolated in the laboratory, and most of these DNA transferring, drug resistant species will only grow at or near 37 degree C, (or only at average body temperature,) g) any cooler temperatures or normal atmospheric oxygen will readily prohibit the proper drug-resistant phase from being cultured and/or otherwise detected; h) they will NOT grow in any normal laboratory where they are routinely exposed to normal atmospheric oxygen, i) and they tend to only grow and reproduce (at normal body temperatures) and their reproductive activities are mostly present, only when oxygen levels are low.
In addition to the evidences that are readily available showing the existence of the transferable packets of DNA by electronic microscope and metal staining techniques, all cancers and tumors can be neatly categorized and otherwise explained in terms their being either: 1) tumors, which usually grow very slowly and result from a DNA packet without any transformation module(s), so they will only replicate within a given, narrow range of activity Â and characteristically produce only some very limited variation of the same cell, over and over again; or 2) neoplastic cancers, the result of a transferable DNA packet that has the DNA modules within it to readily change or transform the DNA, so as to produce variable cell features, and they thereby replicate along a very broad range of activity, which includes the ability to self-transfer, better known as metastasis; and 3) hormone sensitive cancers, which are driven by a transferable DNA packet (as above,) that also contain their own form of normal hormonal features. Hence, the importance of the previous note that some bacteria in the Clostridium family break down fats into the minor estrogens, otherwise known as Âimpeded estrogens.Â In other words, the characteristic features that surround the production of various impeded estrogens in many breast cancers are all explained when the otherwise foreign DNA from a drug-resistant Clostridium gains entry into a breast cell. They will be operational in that area, on a monthly, periodic basis, breaking down the various milk fats that are left among the swollen breast tissue, each time the expected pregnancy does not take place. Hence, once they pass their DNA into a given breast cell that cell becomes cancerous; it rapidly divides, and the every growing mass of cells will produce the minor estrogens forms known mainly as the Âimpeded estrogens.Â
Their growth conditions for this type of bacteria are so extreme, however, that one such cancer causing, DNA transferring bacteria that has already been found, and it turned out to be so unusual that they in some ways approximate the growth conditions of Campylobacter. After more than a year of patient investigation, when researchers had attempted time and time again to isolate one such microorganism, often found to be closely associated with Candida, they only succeeded when cultures were inadvertently left evidently in a sun warmed incubator, or apparently in a non-air conditioned laboratory, for a period of 6 days over the Easter holidays! This chance prolongation of temperatures and the incubation period (from the usual 2 days to 6 days!) resulted in the successful growth and isolation of a microorganism. (see Lynch, Nancy A., ÂHelicobacter pylori and Ulcers: a Paradigm Revised," University of Iowa College of Medicine, Dept of Pathology.) I believe it to also be the organism once described in 1925 in The British Medical Journal, as being Âobtained almost constantly from cancers,Â and that same bacteria is now better known as Helicobacter pylori. In 1925, James Young wrote on it (at a time when stomach cancers were the most active of cancers. . .) ÂOf all the diseases of man cancer has probably the most unique features; it has little direct contact at any point with other morbid processes, and, for that reason, analogy is of small positive help in a search for the cause. The essential fact which marks it off, is that, beginning in the proliferation of a small group of cells, it ends, when running its ordinary course, as a myriad host of anarchic cells destroying by direct inroad the healthy surroundings and burrowing often into the springs of life itself. Any class of cells in the body may be seized with this tragic impetus, and, according to their site of origin, so will the cancer differ in structure and, it may be, in deadliness; but in all, no matter what the origin, the same unique features dominate the process, in all we have a plague of riotous, cells spreading as a blight without control or cessation.Â (Young, James, ÂA New Outlook On Cancer,Â The British Medical Journal, Jan. 10, 1925, pages 60-64.)
Young also wrote one other, highly important note. He stated: ÂOne of the most important positive facts in regard to the etiology of cancer acquired within recent years concerns the role played by chronic irritation. The experimental production of cancer in animals by the exposure of the skin to' chemical irritants, of which there are now several, has lifted a long established clinical observation into the realm of proved laboratory fact. It can now be claimed with certainty that cancer can be induced by factors which vary as greatly in their nature as do tar, soot, arsenic, x rays, heat, etc. By some, these isolated and distinct irritant influences have been adduced as necessarily so many direct ÂcausesÂ of this unique process. To many who have followed the specious arguments which have sometimes been advanced in support of this extraordinary claim it must come as a surprise that even distinguished observers may be negligent of the warning lessons of medical history. Sixty years ago to claim that the specific disease, phthisis (i. e., tuberculosis) of the lungs, had a multiplicity of direct causes was in keeping with - the knowledge of the, period. Thus it was believed that the inflammation of a bronchitis, the irritation of the inhaled fragments of stone with which the mason worked, etc., were each one a direct "cause" of pulmonary phthisis, and that there were many other causes as well-for example, heredity, unhealthy environment, etc. Time has shown that the greatly differing direct irritants and the other indirect agencies operate in one common way - by increasing cell and tissue susceptibility to the one common factor, the tubercle bacillus. In many other cases the history of medical progress has shown that, where several causes have been advanced to account for a disease, these have proved to be nothing more than factors preceding, and predisposing to, the single common agent.Â (Young, James, ÂA New Outlook On Cancer,Â The British Medical Journal, Jan. 10, 1925, page 65.)
Indeed, it is the same story today! When it comes to stomach cancers, there is supposedly a whole list of things that cause ulcers and stomach cancers, and it all turns out to be caused by the transfer of DNA from the drug-resistant Helicobacter pylori bacteria. When tested for their cancer causing properties, researchers used the 20-year old archived blood samples of patients in a large sample who had eventually developed gastric malignancy were tested for antibodies to H. pylori, and then they compared that to samples of an otherwise similar, but cancer free control group. Statistical analysis of the data indicated that those infected with H. pylori at the time the blood was collected were up to six times more likely to have subsequently developed a malignancy. At that point, the connection of H pylori to stomach cancer became so certain that the World Health Organization International Agency for Research in Cancer classified it as a class I carcinogen. (Lynch, Nancy A., ÂHelicobacter pylori and Ulcers: a Paradigm Revised, University of Iowa College of Medicine, Dept of Pathology.) Another way to put it, at one time, before the process of homogenization was invented, stomach cancers were so prevalent that they were the leading cause of death in America. It is fairly obvious at this point that the milk fats in some instances actually contained DNA transferring bacteria from the Clostridium family Â and the transferable DNA from these milk fat bacteria were what was causing the stomach cancers. In other words, stomach cancers narrowed down to being caused only by H. pylori, more than 40 years ago, after homogenization put an end to what was once the most deadly disease in America.
After that, then smoking became the number one killer Â which leads to the conclusion that even this disease is actually caused by a very similar type of DNA transferring, drug resistant bacteria. These bacteria commonly grow and inhabit the major air ways, where it tend to often transfer its DNA into the same type of cells lining certain frequently damaged tissues within the smoker's lungs.
And what about those persons who do not smoke, but they are subjected to second hand smoke? Incredibly, the transferable DNAÂs that causes the lung cancers are so vastly small, that they could be easily carried about or transferred on nothing more than a tiny smoke particle! And what about those smokers who smoke and smoke and smoke, and they seem to never get sick? Those persons are extremely fortunate or just plain lucky in that, there was a time when they too became infected, but their immune system caught it in time, set up the proper antibodies, and they will never be sick with this form of cancer. Are they then, ÂcarriersÂ of the cancer causing drug resistant bacteria? There is every probability that they actually are a Âcarrier,Â just as surely as any person may actually become a ÂcarrierÂ of TB.
In conclusion, just as stomach cancers were discovered to be caused by drug resistant Helicobacter pylori, the fact is that H. pylori IS AN INCREDIBLY DIFFICULT BACTERIA TO ISOLATE AND DECTECT Â YET IT HAS THE ABILITY TO TRANFER DNA - which is known to result in rapidly growing, drug-resistant, stomach malignancies, etc., Hence, this points to the fact that there are, of course, other drug resistant bacteria WITH THIS VERY SAME ABILITY TO TRANSFER DNA,Â which are responsible for the many other cancers and tumors. In other words, to briefly paraphrase what was once observed by James Young, several causes have been advanced to account for the various tissues in which the disease of cancer emerges, and all those have proved to be nothing more than factors preceding, and predisposing to, a single common agent Â the transfer of DNA into a cell from a drug resistant bacteria. Hence, cancers and tumors are caused by the DNA modules (i. e., plasmid DNA) that has become active after being passed into a cell, from a drug-resistant bacteria.Â In brief, this also means that every oncogene now listed by various scientists around the world, is actually composed of one of these little packets of transferable DNAÂs, and that cell-directing material is simply being integrated into a given cell, once that particular cell divides. To be strictly scientific, this explains each oncogene whether it refers to a cancer causing oncogene or not. When the cell becomes cancerous, nowadays the researchers say that the cellÂs genes are Âpotentially oncogenesÂ and the term has thus shifted to protooncogenes. It doesn't matter what itÂs called, simply put, the transfer of drug resistant DNA causes the cells genes to become cancerous Â by causing any of a variety of changes, such as over-expression, under-expression, mutation, inactivation, differing substrates, different affinity for substrate, etc. and so this identifies were all the various oncogenes come from. In other words, every tissue has itsÂ own ecological values - which match those of the environment required by certain strains of the various types of DNA transferring bacteria, and this is the basic cause of cancers, tumors, and each of a long list of other, gene related diseases.
Nov 3, 2003, by Bruce D. McKay
This is all original research by Bruce D. McKay. It is the result of an extensive, thirty year epistemological study of the major features of oncology and many other aspects of the biological sciences. McKay, who just turned 62 year old, has spent most of his adult life as a biological epistemologist. He can be reached for further comment, or other information etc., at:
11308 N. Hamner Ave. in Tampa, FL, 33612. Phone number: (813) 933-4905.
Brucedonaldmckay@aol. com bmckay3@tampabay. rr. com